Corrigendum to “Tepraloxydim metabolite exposure disrupts placental function revealed by single-cell sequencing and metabolomics” [Biochem. Biophys. Res. Commun. 827 (2026) 154073] (Biochemical and Biophysical Research Communications (2026) 827, (S0006291X26008375), (10.1016/j.bbrc.2026.154073))

The authors regret that, due to an oversight, errors occurred during the assembly of Fig. 1B and D. During a subsequent verification of the original materials, we identified these mistakes and have now corrected them. The authors sincerely apologize for these unintentional errors. The corrections do not affect the results, interpretation, or conclusions of the article. We take full responsibility for these mistakes and extend our apologies to the editor, reviewers, and readers for any inconvenience caused. The figure legends remain unchanged from the original article. The corrections are outlined below: A) In Fig. 1B, the originally published super-resolution confocal microscopy panels for both the DMP and NCP groups were incorrectly assembled during figure preparation. The published images have been replaced with the correct representative images from the corresponding experimental groups. B) In Fig. 1D, the incorrectly assembled Raman spectroscopy panel confirming the presence of the active metabolite of Tepraloxydim in placental tissue has been replaced with the correct panel. In addition, we have added a figure legend for Fig. 1E to clarify that it represents the standard curve for microplastic quantification. Fig. 1E has been corrected accordingly: 3.1. Transplacental and Subcellular Localization of DMP in Murine Placenta. To better understand cellular responses of the placenta against the ubiquitous newer fungicide formulations during pregnancy, we exposed time-pregnant mice to DMP established acceptable daily intakes to 180 mg/kg body weight and performed scRNA seq analysis on the transcriptome of the whole mouse placenta including decidua (Fig. 1A). Analysis of the DMP-exposed placentas revealed a substantial accumulation of fluorescent substances, demonstrating the ability of DMP to traverse the blood-placental barrier (Fig. 1B). Super-resolution confocal microscopy revealed that DMP-derived fluorescent signals exhibited a reticular distribution pattern reminiscent of endoplasmic reticulum (ER) morphology (Fig. 1B). Transmission electron microscopy further identified electron-dense deposits within membrane-bound compartments morphologically consistent with ER structures (Fig. 1C). Additionally, Raman spectroscopy confirmed the presence of DMP by detecting their distinctive peaks in the tissue (Fig. 1D). These results collectively demonstrate the efficient transplacental transfer and specific subcellular accumulation of DMP in placental tissue, providing crucial insights into its placental penetration characteristics and potential cellular targets. As shown in Fig. 1E, a standard curve was generated using known concentrations of microplastic standards to establish the relationship between signal intensity and microplastic concentration.[Figure