白藜芦醇通过TGF-β1/Smad3信号通路抑制心肌成纤维细胞中AngⅡ诱导的 Ⅰ/Ⅲ 型胶原合成

Translated title of the contribution: Resveratrol inhibits the expression of typeⅠ and type Ⅲ collagen induced by AngⅡ in cardiac fibroblasts through regulating the TGF-β1/Smad3 signaling pathway
  • Xiao Zhen Zhuo
  • , Yi Liu
  • , Na Li
  • , Jing Jing Yin
  • , Wen Xi
  • , Jun Hui Liu

Research output: Contribution to journalArticlepeer-review

2 Scopus citations

Abstract

Objective: To explore the inhibitory effect of resveratrol on hypertension-related myocardial fibrosis and the key role of TNF-β/Smads signaling pathway in the anti-fibrosis of resveratrol. Methods: The expression of vimentin in the primary rat CFs was evaluated by immunofluorescence to determine the cell type. CFs were treated in different conditions, the mRNA expression levels of type Ⅰ and type Ⅲ collagen and TGF-β1 were detected by Real-time PCR; the protein expression levels of type Ⅰ and type Ⅲ collagen, TGF-β1, Smad-3 and P-Smad-3 were detected by Western blot. The secretion levels of type Ⅰ collagen and TGF-β1 in CFs supernatant were measured by ELISA assay. Results: Rat CFs were successfully extracted, and vimentin expression was obvious. Real-time PCR results indicated that the gene expression levels of collagen Ⅰ and Ⅲ and TGF-β1 in CFs by exposure to AngⅡ were significantly increased as compared with those in normal group (P<0.05). However, AngⅡ-induced collagen Ⅰ and Ⅲ and TGF-β1 mRNA upregulation was inhibited by Res treatment (P<0.05). Western blot analysis showed that the protein expressions of collagen Ⅰ and Ⅲ and TGF-β1 in CFs were also increased after exposed to AngⅡ when compared to the normal controls (P<0.05). Similarly, AngⅡ-mediated collagen Ⅰ and Ⅲ and TGF-β1 upregulation was prevented by Res treatment (P<0.05). In addition, the phosphorylation level of Smad-3 was enhanced by both interventions (P<0.05). However, AngⅡ stimulated TGF-β1 upregulation while Smad-3 phosphorylation was suppressed by Res treatment (P<0.05). The secretion levels of collagen Ⅰ and TGF-β1 in CFs supernatant increased significantly in CFs exposed to AngⅡ condition as compared with those in normal condition (P<0.05). However, AngⅡ increased collagen Ⅰ and TGF-β1 secretion was prevented by Res intervention (P<0.05). Conclusion: Resveratrol inhibits the expressions of type Ⅰ and type Ⅲ collagen induced by AngⅡ in cardiac fibroblasts through regulating the TGF-β1/Smad3 signaling pathway.

Translated title of the contributionResveratrol inhibits the expression of typeⅠ and type Ⅲ collagen induced by AngⅡ in cardiac fibroblasts through regulating the TGF-β1/Smad3 signaling pathway
Original languageChinese (Traditional)
Pages (from-to)525-529 and 557
JournalJournal of Xi'an Jiaotong University (Medical Sciences)
Volume39
Issue number4
DOIs
StatePublished - Jul 2018
Externally publishedYes

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